Metabolic alkalosis
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Etiology
Saline-Responsive (UCl <10 meq/d)Excessive body bicarbonate content
- Renal alkalosis
- Diuretic therapy
- Poorly reabsorbable anion therapy: carbenicillin, penicillin, sulfate, phosphate
- Posthypercapnia
- Gastrointestinal alkalosis
- Loss of HCl from vomiting or nasogastric suction
- Intestinal alkalosis: chloride diarrhea
- Exogenous alkali
- NaHCO3 (baking soda)
- Sodium citrate, lactate, gluconate, acetate
- Transfusions
- Antacids
- Contraction alkalosis
- Renal alkalosis, Normotensive
- Bartter's syndrome (renal salt wasting and secondary hyperaldosteronism)
- Severe potassium depletion
- Refeeding alkalosis
- Hypercalcemia and hypoparathyroidism
- Renal alkalosis, Hypertensive
- Endogenous mineralocorticoids
- Primary aldosteronism
- Hyperreninism
- Adrenal enzyme deficiency: 11- and 17-hydroxylase
- Liddle's syndrome
- Exogenous mineralocorticoids
- European licorice
- Endogenous mineralocorticoids
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Etiology
Saline-Responsive (UCl <10 meq/d)Excessive body bicarbonate content
- Renal alkalosis
- Diuretic therapy
- Poorly reabsorbable anion therapy: carbenicillin, penicillin, sulfate, phosphate
- Posthypercapnia
- Gastrointestinal alkalosis
- Loss of HCl from vomiting or nasogastric suction
- Intestinal alkalosis: chloride diarrhea
- Exogenous alkali
- NaHCO3 (baking soda)
- Sodium citrate, lactate, gluconate, acetate
- Transfusions
- Antacids
- Contraction alkalosis
- Renal alkalosis, Normotensive
- Bartter's syndrome (renal salt wasting and secondary hyperaldosteronism)
- Severe potassium depletion
- Refeeding alkalosis
- Hypercalcemia and hypoparathyroidism
- Renal alkalosis, Hypertensive
- Endogenous mineralocorticoids
- Primary aldosteronism
- Hyperreninism
- Adrenal enzyme deficiency: 11- and 17-hydroxylase
- Liddle's syndrome
- Exogenous mineralocorticoids
- European licorice
- Endogenous mineralocorticoids
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