Seborrheic Dermatitis
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Basics
Description
- A common and chronic papulosquamous inflammatory skin disorder
- Affects all age groups and varies from mild dandruff to extensive adherent scale
- Found in areas with high concentrations of sebaceous follicles and glands
- Sharply demarcated yellow to red to brown, greasy, scaling, crusting patches/plaques
- Periods of remission and exacerbation frequent in adults
Etiology
- Exact pathogenesis not fully understood
- Multifactorial with environmental, genetic, hormonal, immunologic, microbial, and nutritional influences
- Strong association with Malassezia yeasts
- Complex physiologic response:
- Immunologic
- Inflammatory
- Hyperproliferation
- Disease flares are common with physical and emotional stress or illness
- Factors predisposing patients to develop seborrheic dermatitis and more severe or refractory disease:
- Parkinson disease
- Paralysis
- HIV/AIDS
- Mood disorders including depression
- Congestive heart failure
- Immunosuppression in premature infants
- Medications known to induce or aggravate seborrheic dermatitis include:
- Arsenic
- Auranofin
- Aurothioglucose
- Buspirone
- Carbamazepine
- Chlorpromazine
- Cimetidine
- Ethionamide
- Gold
- Griseofulvin
- Haloperidol
- Interferon-α
- Lithium
- Methoxsalen
- Methyldopa
- Phenothiazines
- Phenytoin
- Primidone
- Psoralen
- Stanozolol
- Thiothixene
- Trioxsalen
-- To view the remaining sections of this topic, please log in or purchase a subscription --
Basics
Description
- A common and chronic papulosquamous inflammatory skin disorder
- Affects all age groups and varies from mild dandruff to extensive adherent scale
- Found in areas with high concentrations of sebaceous follicles and glands
- Sharply demarcated yellow to red to brown, greasy, scaling, crusting patches/plaques
- Periods of remission and exacerbation frequent in adults
Etiology
- Exact pathogenesis not fully understood
- Multifactorial with environmental, genetic, hormonal, immunologic, microbial, and nutritional influences
- Strong association with Malassezia yeasts
- Complex physiologic response:
- Immunologic
- Inflammatory
- Hyperproliferation
- Disease flares are common with physical and emotional stress or illness
- Factors predisposing patients to develop seborrheic dermatitis and more severe or refractory disease:
- Parkinson disease
- Paralysis
- HIV/AIDS
- Mood disorders including depression
- Congestive heart failure
- Immunosuppression in premature infants
- Medications known to induce or aggravate seborrheic dermatitis include:
- Arsenic
- Auranofin
- Aurothioglucose
- Buspirone
- Carbamazepine
- Chlorpromazine
- Cimetidine
- Ethionamide
- Gold
- Griseofulvin
- Haloperidol
- Interferon-α
- Lithium
- Methoxsalen
- Methyldopa
- Phenothiazines
- Phenytoin
- Primidone
- Psoralen
- Stanozolol
- Thiothixene
- Trioxsalen
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