Hernias
Basics
Basics
Basics
Description
Description
Description
- Protrusion of a bodily structure or organ through a defect in tissues normally containing it
- Most commonly classified by region (ventral, groin, pelvic, flank)
- Groin:
- Indirect inguinal hernia:
- Protrusion through internal ring due to persistent process vaginalis
- Right side more common than left
- Direct inguinal hernia:
- Protrusion due to weakness or defect in transversalis area in Hesselbach triangle (inguinal ligament inferiorly, inferior epigastric vessels laterally, rectus abdominus medially)
- Femoral hernia:
- Protrusion into femoral canal beneath inguinal ligament
- More common in females than males
- Incarceration frequent due to protrusion through small orifice
- Flank:
- Lumbar hernia:
- Protrusion through superior (more common) or inferior (less common) lumbar triangle of posterior abdominal wall
- Usually middle-aged males
- Left side more common than right
- Pelvic:
- Perineal hernia:
- Protrusion through a congenital (rare) or acquired defect in the pelvic floor
- Acquired perineal hernias associated with history of pelvic operations
- Sciatic hernia:
- Protrusion through the greater or lesser sciatic foramen
- Obturator hernia:
- Protrusion through the obturator canal
- Usually thin elderly females
- Ventral:
- Incisional hernia:
- Protrusion due to breakdown of previous surgical fascial closure
- High recurrence rate (up to 30–40%)
- Epigastric hernia:
- Protrusion at the midline between xiphoid and umbilicus
- Spigelian hernia:
- Protrusion through oblique fascia lateral to rectus abdominis muscle (also known as a lateral ventral hernia)
- Umbilical hernia:
- Protrusion through fibromuscular umbilical ring
- Can be congenital (in children due to failure of umbilical ring to close) or acquired (more common etiology in adults)
Epidemiology
Epidemiology
Epidemiology
- Hernia repair is an extremely common general surgical procedure (>1 million performed in the US annually)
- Prevalence: 10% of population:
- 75% inguinal (50% direct, 25% indirect)
- 25–30 % ventral
- 3–5% femoral
Etiology
Etiology
Etiology
- Congenital: Defect in the abdominal wall is present from birth
- Acquired: Defect in the abdominal wall develops due to weakening or disruption of fibromuscular tissues
- Any increased intra-abdominal pressure can lead to hernia (obesity, heavy lifting, coughing, straining, organomegaly, intra-abdominal masses, ascites, peritoneal dialysis)
- Reducible hernia:
- Protruding structures can be returned to abdominal cavity
- Incarcerated hernia:
- Contents of hernia cannot be manipulated back into abdominal cavity
- Strangulated hernia:
- Vascular compromise of entrapped bowel contained within hernia leading to ischemia and gangrene (skin color changes may be apparent)
- Higher risk in hernias with small openings and large sacs
- Signs and symptoms of bowel obstruction or ischemia may occur (nausea/vomiting, fever, leukocytosis)
- Richter hernia: Only the antimesenteric portion of the bowel wall protrudes into the hernia defect:
- Bowel may not be obstructed but can rapidly become strangled and gangrenous
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